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drug resistance gene detection kit for m. tuberculosis  (CapitalBio Corporation)

 
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    CapitalBio Corporation drug resistance gene detection kit for m. tuberculosis
    Drug Resistance Gene Detection Kit For M. Tuberculosis, supplied by CapitalBio Corporation, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/m%2E+tuberculosis+drug+resistance+gene+detection+kit/drug+resistance+detection+kit/10__1016_slash_j__jallcom__2021__162202-77-36-38
    Average 90 stars, based on 1 article reviews
    drug resistance gene detection kit for m. tuberculosis - by Bioz Stars, 2026-09
    90/100 stars

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    Related Articles

    Microarray:

    Article Title: Prevalence and molecular characteristics of drug-resistant Mycobacterium tuberculosis in Hainan, China: from 2014 to 2019
    Article Snippet: This study was based on M. tuberculosis drug resistance gene detection kit (CapitalBioTM DNA microarray method, Beijing CapitalBio Technology, 301,035), which can specifically detect the mutations of rpoB , katG and inhA .

    Comparison:

    Article Title: Prevalence and molecular characteristics of drug-resistant Mycobacterium tuberculosis in Hainan, China: from 2014 to 2019
    Article Snippet: This study was based on M. tuberculosis drug resistance gene detection kit (CapitalBioTM DNA microarray method, Beijing CapitalBio Technology, 301,035), which can specifically detect the mutations of rpoB , katG and inhA .

    Mutagenesis:

    Article Title: Prevalence and molecular characteristics of drug-resistant Mycobacterium tuberculosis in Hainan, China: from 2014 to 2019
    Article Snippet: This study was based on M. tuberculosis drug resistance gene detection kit (CapitalBioTM DNA microarray method, Beijing CapitalBio Technology, 301,035), which can specifically detect the mutations of rpoB , katG and inhA .



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    drug resistance gene detection kit for m. tuberculosis  (CapitalBio Corporation)
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    Imaging examination of pulmonary <t>tuberculosis.</t> CT scans revealed lobular and nodular high-density shadows, with cavity shadows observed in some lesions.
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    Image Search Results


    Imaging examination of pulmonary tuberculosis. CT scans revealed lobular and nodular high-density shadows, with cavity shadows observed in some lesions.

    Journal: Frontiers in Microbiology

    Article Title: Unique pathological features and drug resistance patterns in cutaneous tuberculosis

    doi: 10.3389/fmicb.2025.1574051

    Figure Lengend Snippet: Imaging examination of pulmonary tuberculosis. CT scans revealed lobular and nodular high-density shadows, with cavity shadows observed in some lesions.

    Article Snippet: Drug resistance testing used the M. tuberculosis drug resistance mutation gene detection kit (Yaneng Biotechnology Co., Ltd., Shenzhen, China).

    Techniques: Imaging

    The typical pathological manifestations of pulmonary tuberculosis were chronic granulomatous inflammation (H&E-1) with caseous necrosis (H&E-2). H&E staining showed coagulation necrosis (H&E-3), inflammatory necrosis (H&E-4), mucous degeneration (H&E-5), fibroplasia (H&E-6), hemorrhage (H&E-7), acute inflammation (H&E-8), abscess (H&E-9), granulation tissue formation (H&E-10). Acid-fast staining showed M. tuberculosis . Immunohistochemical staining was used to label granuloma (CD68-1), alveolar epithelial cells (CK-1), proliferation rate (Ki-67-1) and lymphocyte infiltration (LCA-1) in tuberculosis lesions. Immunohistochemical staining was also used to label macrophage (CD68-2, CD68-3), alveolar epithelial cells (CK-2) and lymphocyte infiltration (LCA-2) in the surrounding normal lung tissue.

    Journal: Frontiers in Microbiology

    Article Title: Unique pathological features and drug resistance patterns in cutaneous tuberculosis

    doi: 10.3389/fmicb.2025.1574051

    Figure Lengend Snippet: The typical pathological manifestations of pulmonary tuberculosis were chronic granulomatous inflammation (H&E-1) with caseous necrosis (H&E-2). H&E staining showed coagulation necrosis (H&E-3), inflammatory necrosis (H&E-4), mucous degeneration (H&E-5), fibroplasia (H&E-6), hemorrhage (H&E-7), acute inflammation (H&E-8), abscess (H&E-9), granulation tissue formation (H&E-10). Acid-fast staining showed M. tuberculosis . Immunohistochemical staining was used to label granuloma (CD68-1), alveolar epithelial cells (CK-1), proliferation rate (Ki-67-1) and lymphocyte infiltration (LCA-1) in tuberculosis lesions. Immunohistochemical staining was also used to label macrophage (CD68-2, CD68-3), alveolar epithelial cells (CK-2) and lymphocyte infiltration (LCA-2) in the surrounding normal lung tissue.

    Article Snippet: Drug resistance testing used the M. tuberculosis drug resistance mutation gene detection kit (Yaneng Biotechnology Co., Ltd., Shenzhen, China).

    Techniques: Staining, Coagulation, Immunohistochemical staining

    The typical pathological manifestations of CTB were chronic granulomatous inflammation (H&E-1) with caseous necrosis (H&E-2). H&E staining showed coagulation necrosis (H&E-3), inflammatory necrosis (H&E-4), exudation (H&E-5), fibroplasia (H&E-6), hemorrhage (H&E-7), granulation tissue formation (H&E-8), inflammatory necrosis (H&E-9). Acid-fast staining showed M. tuberculosis . Immunohistochemical staining was used to label granuloma (CD68-1), proliferation rate (Ki-67), lymphocyte (LCA-1) and T cell (CD3) infiltration in tuberculosis lesions. Immunohistochemical staining was also used to label macrophage (CD68-2), and lymphocyte infiltration (LCA-2) in the surrounding normal skin tissue.

    Journal: Frontiers in Microbiology

    Article Title: Unique pathological features and drug resistance patterns in cutaneous tuberculosis

    doi: 10.3389/fmicb.2025.1574051

    Figure Lengend Snippet: The typical pathological manifestations of CTB were chronic granulomatous inflammation (H&E-1) with caseous necrosis (H&E-2). H&E staining showed coagulation necrosis (H&E-3), inflammatory necrosis (H&E-4), exudation (H&E-5), fibroplasia (H&E-6), hemorrhage (H&E-7), granulation tissue formation (H&E-8), inflammatory necrosis (H&E-9). Acid-fast staining showed M. tuberculosis . Immunohistochemical staining was used to label granuloma (CD68-1), proliferation rate (Ki-67), lymphocyte (LCA-1) and T cell (CD3) infiltration in tuberculosis lesions. Immunohistochemical staining was also used to label macrophage (CD68-2), and lymphocyte infiltration (LCA-2) in the surrounding normal skin tissue.

    Article Snippet: Drug resistance testing used the M. tuberculosis drug resistance mutation gene detection kit (Yaneng Biotechnology Co., Ltd., Shenzhen, China).

    Techniques: Staining, Coagulation, Immunohistochemical staining

    Evaluation and comparison of drug resistance rate of anti- tuberculosis drugs in 2014–2019

    Journal: BMC Microbiology

    Article Title: Prevalence and molecular characteristics of drug-resistant Mycobacterium tuberculosis in Hainan, China: from 2014 to 2019

    doi: 10.1186/s12866-021-02246-7

    Figure Lengend Snippet: Evaluation and comparison of drug resistance rate of anti- tuberculosis drugs in 2014–2019

    Article Snippet: This study was based on M. tuberculosis drug resistance gene detection kit (CapitalBioTM DNA microarray method, Beijing CapitalBio Technology, 301,035), which can specifically detect the mutations of rpoB , katG and inhA .

    Techniques: Comparison

    Trends of different drug-resistance patterns among 1484 culture-confirmed TB cases in Hainan, 2014 to 2019. In new cases, for INH resistance (χ 2 = 2.813, P = 0.729); for RIF resistance (χ 2 = 3.181, P = 0.672); for MDR-TB (χ 2 = 4.210, P = 0.520); for XDR-TB (χ 2 = 4.383, P = 0.496). In retreatment cases, for INH resistance (χ 2 = 9.512, P = 0.090); for RIF resistance (χ 2 = 14.257, P = 0.014); for MDR-TB (χ 2 = 10.328, P = 0.066); for XDR-TB (χ 2 = 7.670, P = 0.175). Note: *new cases, **retreatment cases. Abbreviation: INH-R, isoniazid resistance; RIF-R, rifampin resistance; MDR-TB, multidrug resistant tuberculosis; XDR-TB, extensively drug resistant tuberculosis

    Journal: BMC Microbiology

    Article Title: Prevalence and molecular characteristics of drug-resistant Mycobacterium tuberculosis in Hainan, China: from 2014 to 2019

    doi: 10.1186/s12866-021-02246-7

    Figure Lengend Snippet: Trends of different drug-resistance patterns among 1484 culture-confirmed TB cases in Hainan, 2014 to 2019. In new cases, for INH resistance (χ 2 = 2.813, P = 0.729); for RIF resistance (χ 2 = 3.181, P = 0.672); for MDR-TB (χ 2 = 4.210, P = 0.520); for XDR-TB (χ 2 = 4.383, P = 0.496). In retreatment cases, for INH resistance (χ 2 = 9.512, P = 0.090); for RIF resistance (χ 2 = 14.257, P = 0.014); for MDR-TB (χ 2 = 10.328, P = 0.066); for XDR-TB (χ 2 = 7.670, P = 0.175). Note: *new cases, **retreatment cases. Abbreviation: INH-R, isoniazid resistance; RIF-R, rifampin resistance; MDR-TB, multidrug resistant tuberculosis; XDR-TB, extensively drug resistant tuberculosis

    Article Snippet: This study was based on M. tuberculosis drug resistance gene detection kit (CapitalBioTM DNA microarray method, Beijing CapitalBio Technology, 301,035), which can specifically detect the mutations of rpoB , katG and inhA .

    Techniques:

    Evolution of drug resistance mutation sites of Mycobacterium tuberculosis to first-line anti tuberculosis drugs isoniazid and rifampin in 2014–2019

    Journal: BMC Microbiology

    Article Title: Prevalence and molecular characteristics of drug-resistant Mycobacterium tuberculosis in Hainan, China: from 2014 to 2019

    doi: 10.1186/s12866-021-02246-7

    Figure Lengend Snippet: Evolution of drug resistance mutation sites of Mycobacterium tuberculosis to first-line anti tuberculosis drugs isoniazid and rifampin in 2014–2019

    Article Snippet: This study was based on M. tuberculosis drug resistance gene detection kit (CapitalBioTM DNA microarray method, Beijing CapitalBio Technology, 301,035), which can specifically detect the mutations of rpoB , katG and inhA .

    Techniques: Mutagenesis